The one thing that I know eating meat (and/or other tyrosine sources) affects, is the switching frequency of tyrosine kinases in your cells. (Helpful mental model: some of your cellular organelles are finite-state machines. Tyrosine kinases trigger their state transitions.)
And I don’t know with the same level of certainty, but do believe, that this switching/state-transitioning in your cells is a major contributor to cellular senescence. Decreasing the switching frequency of tyrosine kinases in the body is, IIRC, being proposed as as both 1. a major reason that intermittent fasting increases lifespan, and 2. as a potential mechanism by which some aquatic species that live in extreme cold can live for hundreds of years.
It’d be nice to be able to turn this effect down, is what I’m saying. Sadly, it seems that doing so makes some things (like maintaining homeostasis during exercise) much harder. So there’s, maybe, a trade-off here: a longer life, but one constrained to only a part of the range of human activity. Interesting choice.
And I don’t know with the same level of certainty, but do believe, that this switching/state-transitioning in your cells is a major contributor to cellular senescence. Decreasing the switching frequency of tyrosine kinases in the body is, IIRC, being proposed as as both 1. a major reason that intermittent fasting increases lifespan, and 2. as a potential mechanism by which some aquatic species that live in extreme cold can live for hundreds of years.
It’d be nice to be able to turn this effect down, is what I’m saying. Sadly, it seems that doing so makes some things (like maintaining homeostasis during exercise) much harder. So there’s, maybe, a trade-off here: a longer life, but one constrained to only a part of the range of human activity. Interesting choice.