When you use EcoMode with them you only lose ~5% performance, but are still ~30% ahead of the corresponding 5000-series CPU. You can reduce PPT/TDP even further while still ahead.
Definetly not that one if you plan to pair with a dedicated GPU! The 5700X has twice the L3 cache. All Ryzen 5000 with a GPU have only 16MB, 5700 has the GPU deactivated.
I have a lower-end Radeon GPU (Navi 24 [Radeon RX 6400]). Which CPU would you suggest with it? I only ever want to use the GPU though, not the CPU's integrated one. I kind of want to get a motherboard that is compatible with the latest AM socket, which is AM5, right? So if I want a CPU with AM5, what would you suggest for the CPU?
But see, this is why it is so difficult. I would have never guessed. I would have to research this A LOT, which I am fine with, but you know.
Bitwig too. Pain point for me is hardware support. My Native Instruments interface works great, due to the in kernel drivers supplied by NI. My other one with loads of IO sadly won't.
People believe what they want. Ergo they side with the people who state that those beliefs are true, makes them feel good. Listening to experts who know better and oppose the held beliefs makes the believers feel bad, ergo they won't believe them.
Sometimes it is good to disregard the opinion of experts who are absolutely sure something can't be done, might by a prerequisite to making it happen.
The four minute mile comes to mind.
Beliefs are powerful, they can enable you to reach goals, become prisons of the mind trapping you or become delusions when feedback is disregarded.
I posted elsewhere in the thread about my late diagnosis. When I was much younger than when I was diagnosed, I was on Bupropion for smoking cessation and it had a pretty significant positive effect for me with respect to my ADHD symptoms (which I didn’t recognize as ADHD at the time). Definitely worth exploring; it doesn’t work for everyone but it can be quite effective if it does work for you.
I would second your suggestion to investigate bupropion. More than a decade ago I was suffering from severe depression. Psychiatrists with meds were an easy-to-access front line treatment, but I was very concerned about SSRI side effects, particularly erectile side effects. It didn't take much persuading to have the psychiatrist prescribe bupropion instead of an SSRI. I too felt it helped with my then undiagnosed ADHD a lot.
As a note, bupropion did make me feel a little funny. It wasn't bad, weird, or intolerable, but yes I did have a faint perceptual awareness that I was in a medicated state.
For the first couple of weeks the first time I took it, Bupropion:
- made me feel pretty energetic. Not buzzed, but maybe counteracted low-grade depression. I was on it because I wanted to quit smoking, because I was hoping quitting smoking would make me feel better/happier
- slightly affected my vision/perception. Colours seemed more vibrant. Everything just felt perceptually… brighter.
- made me really horny, but not in a compulsive way. It wasn’t distracting and didn’t cause any problems, but I definitely had more drive. When it was go time, it was Go Time :)
What's up with the bullet points? This almost reads like a ChatGPT/AI comment, which is especially weird for a personal recollection. Are people writing like frickin robots now?
They’re just lucky I didn’t have a big run-on sentence, like I used to before taking a serious look at how ADHD affected my writing and thought processes, including significant use of parentheticals (because every thought comes with a couple of extra thoughts, for free!)
I have to say, buproprion had severe, life-threatening side effects for me. I'm not in a typically high-risk group for psychotic symptoms, but it caused me to become quite agitated and delusional.
It feels strange to type that now, and I know these side effects are quite rare, but I can't help but warn people when I see it mentioned. It was genuinely the most terrifying experience of my life.
It happened about 6 weeks into treatment, quite acutely. I wasn't even aware it was happening.
Have used them for a long time, never paid for them. GLS is a great bank, happy customer. The crappy ones like to keep your money in their books as long as possible.
> the only way to even approach that would be to have liquid cooling loop ports out the back that you had to plug into an under-desk cooling loop and I don't think anyone is doing that
It is (maybe was) done by XMG and Schenker. Called Oasis IIRC. Yep
It is wrong to claim that ADHD is exclusively hereditary. Also, epigenetics show that behavior changes what is inherited. This is a chance and a responsibility. [1]
More broadly speaking, anything that is claimed to be "hereditary" (which for ADHD the scientific consensus is that it is not exclusively but estimated at around 70%) could also be early nuture and prenatal environment. The way genetic inheritance is researched (identical twins vs. fraternal twins) cannot account for this. [2]
[1] e.g.
"ADHD has been clearly linked with numerous environmental risk factors, particularly around the prenatal and perinatal period. Some of the most robust risk factors identified are maternal prenatal health conditions and psychological distress (e.g. hypertension, obesity, pre-eclampsia, immune activation), in utero exposure to poor diet (with critical factors still being determined), teratogenic effects of certain medications (e.g. acetaminophen) and environmental exposures (e.g. lead), as well as neonatal factors such as prematurity and low birth weight [27]. Other extreme exposures in the postnatal environment (such as extreme infant emotional neglect) have also been associated with an ADHD syndrome [28, 29]."
Cecil, C. A. M., & Nigg, J. T. (2022). Epigenetics and ADHD: Reflections on Current Knowledge, Research Priorities and Translational Potential. Molecular diagnosis & therapy, 26(6), 581–606. https://doi.org/10.1007/s40291-022-00609-y
"The convincing evidence for genes as risk factors for ADHD does not exclude the environment as a source of etiology. The fact that twin estimates of heritability are less than 100% asserts quite strongly that environmental factors must be involved. ADHD’s heritability is high, and that estimate encompasses gene by environment interaction. Thus, it is possible that such interactions will account for much of ADHD’s etiology. Environmental risk factors likely work through epigenetic mechanisms, which have barely been studied in ADHD [148]. The importance of the environment can also be seen in the fact that, as for other complex genetic disorders, much of ADHD’s heritability is explained by SNPs in regulatory regions rather than coding regions [149]."
Faraone, S. V., & Larsson, H. (2019). Genetics of attention deficit hyperactivity disorder. Molecular psychiatry, 24(4), 562–575. https://doi.org/10.1038/s41380-018-0070-0
"Parental stress and parental depression were significantly associated with increased risk for ADHD overall and both symptoms and diagnosis. Specifically, maternal stress and anxiety, maternal prenatal stress, maternal depression, maternal post-partum depression, and paternal depression were positively associated with ADHD. In addition, parental depression was associated with symptoms of ADHD inattentive and hyperactive/impulsive subtypes. Parental antisocial personality disorder was also positively associated with ADHD overall and specifically ADHD diagnosis. Prenatal antidepressant usage was associated with ADHD when measured dichotomously only. These findings raise the possibility that prevention strategies promoting parental mental health and addressing parental stress could have the potential for positive long-term impacts on child health, well-being, and behavioral outcomes."
Robinson, L. R., Bitsko, R. H., O'Masta, B., Holbrook, J. R., Ko, J., Barry, C. M., Maher, B., Cerles, A., Saadeh, K., MacMillan, L., Mahmooth, Z., Bloomfield, J., Rush, M., & Kaminski, J. W. (2024). A Systematic Review and Meta-analysis of Parental Depression, Antidepressant Usage, Antisocial Personality Disorder, and Stress and Anxiety as Risk Factors for Attention-Deficit/Hyperactivity Disorder (ADHD) in Children. Prevention science : the official journal of the Society for Prevention Research, 25(Suppl 2), 272–290. https://doi.org/10.1007/s11121-022-01383-3
[2]
"twin studies fail to separate the effects of genes and the prenatal environment. This failure casts doubt on claims of the relative effects of genes and environment on intelligence, psychiatric disorders, personality and other psychological variables, and other conditions."
"Although many twin studies have been conducted (which is quite an understatement; there are almost
9,000 hits for “twin study” on PubMed!), there have long been critics who argue that they are scientifically
worthless."
"Because heritability is defined by both genetic and environmental influences, it is not a fixed characteristic of a disease or trait, but a population-specific estimate, analogous to, for example, the mean height, cholesterol level or life expectancy in a population. It also cannot be interpretated at the family or individual level."
Kaprio J. (2012). Twins and the mystery of missing heritability: the contribution of gene-environment interactions. Journal of internal medicine, 272(5), 440–448. https://doi.org/10.1111/j.1365-2796.2012.02587.x
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